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The major ingredient in VigRX Plus is Bioperine, a spinoff of black pepper extract, which is known for its ability to boost the physique's absorption of nutrients. This makes the opposite components within the supplement stronger and efficient, including Asian pink ginseng, saw palmetto, and hawthorn berry, all of which have been used for lots of of years in traditional medicine to improve male sexual function.
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VigRX Plus is a well-liked male enhancement complement that has gained significant consideration and reward in current times. Unlike other products in the market, this supplement has been formulated with all-natural elements that work together to supply men with a secure and efficient resolution for his or her sexual well being.
It is indicated, in combination with other approved anticancer drugs, for remission induction in acute nonlymphocytic leukemia (myelogenous, monocytic, erythroid) of adults and for remission induction in acute lymphocytic leukemia in children and adults. It is in the same antineoplastic subclass of anthracyclines as doxorubicin, epirubicin, idarubicin, and valrubicin (1). In animal reproduction studies, exposure to daunorubicin during pregnancy produced teratogenic and toxic effects in rabbits, rats, and mice (1). Restricted fetal and neonatal growth was observed in mice after maternal administration of daunorubicin (1). The use of daunorubicin during pregnancy has been reported in 29 patients, four during the 1st trimester (219). No congenital defects were observed in the 22 (one set of twins) liveborns, but one of these infants was anemic and hypoglycemic and had multiple serum electrolyte abnormalities (8). Severe, transient, druginduced bone marrow hypoplasia occurred in one newborn after in utero exposure to daunorubicin and five other antineoplastic agents (18). The myelosuppression may have been secondary to mercaptopurine, but bone marrow suppression also is a toxic effect of daunorubicin. Results of the remaining pregnancies were three elective abortions (one with enlarged spleen), three intrauterine deaths (one probably due to severe pregnancy-induced hypertension), one stillborn with diffuse myocardial necrosis, and one maternal death (8,9,15,17). Thirteen of the infants (including one set of twins) were studied for periods ranging from 6 months to 9 years and all showed normal growth and development (4,911,14,15,1719). Data from one review indicated that 40% of the infants exposed to anticancer drugs were of low birth weight (20). Except for the infants noted above, long-term studies of growth and mental development in offspring exposed to daunorubicin during the 2nd trimester, the period of neuroblast multiplication, have not been conducted (21). In one report, the use of daunorubicin and other antineoplastic drugs in two males was thought to be associated with congenital defects in their offspring (22). The defects observed were tetralogy of Fallot and syndactyly of the first and second digits of the right foot, and an anencephalic stillborn. Although the authors speculated that the drugs damaged the germ cells without producing infertility and thus were responsible for the defects, any relationship to paternal use of daunorubicin is doubtful due to the lack of experimental evidence and other confirming reports. In a third male, fertilization occurred during treatment with daunorubicin and resulted in the birth of a healthy infant (23). Successful pregnancies have also been reported in two women after treatment with daunorubicin (24). Chromosomal aberrations were observed in the fetus of a 34-year-old woman with acute lymphoblastic leukemia who was treated with multiple antineoplastic agents (12). The clinical significance of these findings is unknown, but since these abnormalities may persist for several years, the potential existed for an increased risk of cancer as well as for a risk of genetic damage in the next generation (12). A position statement from the National Study Commission on Cytotoxic Exposure and a research article involving some antineoplastic agents are presented in the monograph for cyclophosphamide (see Cyclophosphamide). Because of the potential for severe toxicity in a nursing infant, the use of this agent is contraindicated during breastfeeding. Acute leukemia in pregnancy: transient neonatal myelosuppression after combination chemotherapy in the mother.
VigRX Plus 60 caps
Exposure to , hydroxyurea during pregnancy in sickle-Я thalassemia: a report of a case. Influence of chemotherapy for gestational trophoblastic disease on subsequent pregnancy outcome. Polycythemia vera and pregnancy: a case report with the use of hydroxyurea in the first trimester. Although a surveillance study found an increased risk of oral clefts, this defect has not been observed in other studies. The drug is teratogenic in mice and rats, but not in rabbits, at high doses (15). One report suggested that hydroxyzine teratogenicity was mediated by a metabolite (norchlorcyclizine) that was common to four antihistamines (hydroxyzine, buclizine, meclizine, and chlorcyclizine) (3). Highdose hydroxyzine (612 mg/kg/day) resulted in abortions in rhesus monkeys (6). The manufacturer considers hydroxyzine to be contraindicated in early pregnancy because of the lack of clinical data (1,2). The molecular weight of the free base (about 376) suggests that the drug will cross to the embryo and fetus. In 100 patients treated in the 1st trimester with oral hydroxyzine (50 mg daily) for nausea and vomiting, no significant difference from nontreated controls was found in fetal wastage or anomalies (7). A woman treated with 60 mg/day of hydroxyzine during the 3rd trimester gave birth to a normal infant (8). The Collaborative Perinatal Project monitored 50,282 motherchild pairs, 50 of whom had 1st trimester exposure to hydroxyzine (9, pp. Based on five malformed children, a possible relationship was found between 1st trimester use and congenital defects. In a surveillance study of Michigan Medicaid recipients involving 229,101 completed pregnancies conducted between 1985 and 1992, 828 newborns had been exposed to hydroxyzine during the 1st trimester (F. Specific data were available for six defect categories, including (observed/expected) 9/8 cardiovascular defects, 1/0. Withdrawal in a newborn exposed to hydroxyzine 600 mg/day throughout gestation has been reported (10). The mother, who was being treated for severe eczema and asthma, was also treated with phenobarbital, 240 mg/day for 4 days and then 60 mg/day, for mild preeclampsia during the 3-week period before delivery. Symptoms in the newborn, some beginning 15 minutes after birth, consisted of a shrill cry, jitteriness with clonic movements of the upper extremities, irritability, and poor feeding. The presumed drug-induced withdrawal persisted for approximately 4 weeks and finally resolved completely after 2 weeks of therapy with phenobarbital and methscopolamine. Although phenobarbital withdrawal could not be excluded, and neonatal withdrawal is a well-known complication of phenobarbital pregnancy use, the author concluded the symptoms in the infant were primarily caused by hydroxyzine (10). A 1996 report described the use of hydroxyzine, droperidol, diphenhydramine, and metoclopramide in 80 women with hyperemesis gravidarum (11).
